Download Atlas of Heart Diseases: Hypertension: Mechanisms and by Alan B. Weder (auth.), Norman K. Hollenberg MD, PhD (eds.) PDF
By Alan B. Weder (auth.), Norman K. Hollenberg MD, PhD (eds.)
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Extra resources for Atlas of Heart Diseases: Hypertension: Mechanisms and Therapy
Example text
Dallas: American Heart Association; 1993: 36-37. 36. Blaustein M: Sodium ions, calcium ions, blood pressure regulation, and hypertension: a reassessment and a hypothesis. Am JPhysiol1977, 232:C165-C173. 37. Haddy FJ: Potassium, Na+-K+ pump inhibitor and low-renin hypertension. Clin Invest Med 1987, 10:547-554. 38. : Increased lithiumsodium countertransport in red cells of patients with essential hypertenSion. N Engl JMed 1980,302:772-776. 39. Camussi A, Bianchi G: Genetics of essential hypertension: from the unimodal-bimodal controversy to molecular technology.
8] elegantly demonstrated the effect of "mental stress" on blood pressure. Continuous intraarterial blood pressure monitoring was performed on resting patients. When the physician entered the room the blood pressure began to rise and continued to increase when he measured the blood pressure. There was a marked rise in systolic and diastolic blood pressure in both normotensive and hypertensive patients that was associated with an increase in pulse rate (but not to the same extent). The peak blood pressure elevation was recorded 4 minutes after the physician entered the room and slowly decreased over time, so that the blood pressure had almost completely returned to the baseline level after 10 minutes.
J Cardiovasc Pharmacol1988, 12(suppI3):92-98. YPERTENSION: MECHANISMS AND THERAPY 32 41. Weder AB: Red-cell lithium-sodium countertransport and renal lithium clearance in hypertension. N Engl J Med 1986, 314:198-201. 42. : Red blood cell lithiumsodium countertransport in non-modulating essential hypertension. Hypertension 1989, 13:721-726. 43. : Insulin resistance is associated with high sodium-lithium countertransport in essential hypertension. Am J Physiol1991, 261:E684-E691. 44. : Exclusion of the Na+-H+ antiporter as a candidate gene in human essential hypertension.