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By Lamont
This updated reference offers entire discussions on the entire significant infections present in the gastrointestinal tract;emphasizing fresh advances in epidemiology, pathogenesis, and laboratory prognosis and furnishing specified insurance of medical administration.
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Four organic anion transporter proteins (OATPs) and organic anion transporter-2 (OAT2) and organic cation transporter proteins (OCT1) control uptake of anions and cations along the basolateral border (red). Sodium-dependent bile salt uptake occurs via sodium taurocholate protein (NTCP). Five multidrug resistance-associated proteins (MRPs) control secretion (reflux) from the hepatocyte back into space of Disse (blue). After intracellular transit, solute secretion into bile takes place along the canalicular domain through MRP2, multidrugresistant-1 p-glycoprotein (MDR1 and MDR3), bile salt export pump (BSEP), breast cancerresistance protein (BCRP), and flippases (ABCG5/ABCG8).
Leemans R, Manson W, Snijder JAM, Smit JW, Klasen HJ, The TH, Timens W. Immune response capacity after human splenic autotransplantation. Restoration of response to individual pneumococcal vaccine subtypes. Ann Surg 1999;229:279–285 Imaging Agents 3 Historical evolution The introduction of radiocolloids in the 1940s, whose rate of clearance from the circulation was used as an indicator of liver function, gave birth to nuclear hepatology [1]. Imaging of the liver morphology began in 1954 with gold-198 colloid using an automated rectilinear scanner developed by Cassen [2, 3].
Cholangiocytes absorb bile salts through apical sodium-dependent bile salt transporter (ASBT) and OATP1A2. After their uptake, bile salts are excreted through the basolateral membrane (via MRP3) into the peribiliary plexus where they reach the portal circulation. Bile acids and sterols also use organic solute transporters (OSTa/OSTb) to be secreted into the peribiliary plexus through the basolateral domain. Unlike the basolateral border of the hepatocyte, which lacks MRP2, the basolateral border of the cholangiocyte contains MRP2 that controls excretion of organic anions into peribiliary plexus (Fig.