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By Irwin M Arias; et al
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Am J Physiol Gastrointest Liver Physiol , 272, G289–97. , Braet, F. et al. (1994) Cell biology of endothelial and Kupffer cells. Gut , 35, 1509–16. Martinez-Hernandez, A. S. (1993) The hepatic extracellular matrix. I. Components and distribution in normal liver. Virchows Arch A, 423, 1–11. , Somasundaram, R. et al. (1998) Collagens in the liver extracellular matrix bind hepatocyte growth factor. Gastroenterology, 114, 139–52. Smedsrød, B. (2004) Clearance function of scavenger endothelial cells.
All hepatocytes (including polyploid cells) can proliferate, obviating the possibility that functional heterogeneity results from their age-dependent differentiation and ultimate senescence. 11 TURNOVER OF LIVER CELLS All liver parenchymal cells have a finite lifespan which can be shortened by physiological or pathological conditions that increase cell death and cell birth. 025% of which typically will be engaged in DNA synthesis [83]. Cholangiocytes undergo turnover at similar rates. Analysis of the specific lifespans of the other cells of the liver parenchyma is complicated by the fact that their number can be augmented both by local proliferation of cells resident in the liver populations [84] and by repletion from the bone marrow.
45. 46. 47. 48. 49. 50. 51. 52. 53. 54. 55. 56. 57. 58. 59. 60. THE LIVER: REFERENCES subcapsular region in rat livers. Arch Histol Cytol , 66, 239–52. Couinand, C. (1999) Liver anatomy: portal (and suprahepatic) or biliary segmentation. Dig Surg, 16, 459–67. W. V. (1987) Conceptual view of the hepatic vascular bed. Hepatology, 7, 952–63. A. (1963) A linear method for determining the liver sinusoidal and extravascular volumes. J Physiol , 204, 626–40. F. W. (1960) Direct measurement of blood pressure in the minute vessels of the liver.